Pathology Presents: Using pluripotent stem cells to develop models, dissect pathways, and discover new therapeutics

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Speaker

Jessica Young, PhD
Assistant Professor
Department of Pathology
University of Washington

Faculty Sponsor

TBA


Date & Time

September 28, 2016 at 4:30pm - 5:30pm

Location

Health Sciences Building, T-739

Calendar

Pathology Presents

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Add to Calendar 09/28/2016 04:30 PM 09/28/2016 05:30 PM America/Los_Angeles Pathology Presents: Using pluripotent stem cells to develop models, dissect pathways, and discover new therapeutics Pathology Presents: Using pluripotent stem cells to develop models, dissect pathways, and discover new therapeutics

Jessica Young, PhD
Assistant Professor
Department of Pathology
University of Washington
Why Attend? Using pluripotent stem cells to develop models, dissect pathways, and discover new therapeutics for sporadic Alzheimer’s disease Late-onset sporadic Alzheimer’s disease (SAD) is the most common cause of neurodegeneration in the elderly and a growing public health issue. Our lab is working to use patient stem cell-derived neurons to develop an accurate cellular model of SAD. With this model, we will investigate how genes that are associated with SAD risk in population studies impact cellular mechanisms leading to disease. I will give an overview of the projects being initiated in our laboratory, which will focus on the role of the endocytic network in AD pathogenesis. 
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Description

Why Attend?

Using pluripotent stem cells to develop models, dissect pathways, and discover new therapeutics for sporadic Alzheimer’s disease

Late-onset sporadic Alzheimer’s disease (SAD) is the most common cause of neurodegeneration in the elderly and a growing public health issue. Our lab is working to use patient stem cell-derived neurons to develop an accurate cellular model of SAD. With this model, we will investigate how genes that are associated with SAD risk in population studies impact cellular mechanisms leading to disease. I will give an overview of the projects being initiated in our laboratory, which will focus on the role of the endocytic network in AD pathogenesis.